First, I’d like to point out that biochemical theory about the micro-processes taking place during COVID19 infection, and about those almost identical processes taking place during the human cellular and immune system response to COVID19 vaccines, suggests that any of the effective non-pharmaceutical therapies already being used by many clinicians (and citizens) to minimize COVID19 mortality should also help to minimize potential negative effects from the novel RNA and DNA COVID19 vaccines. While the references provided in the legends of each illustration below go over the biochemical theory concerned in very considerable detail, this theory can also be generally gleaned just by carefully examining and comparing Posting Figures 1 and 2.
Next, note in Posting Figure 1 that COVID19 spike proteins produced by introduction of mRNA or DNA vaccines eventually inactivate the same ACE2 receptor entry points on the outside of human cells as do COVID19 spike proteins. Because ACE2 receptors located on the outside of cell have, among other things, a very important role in determining the general balance of the inflammatory/anti-inflammatory processes in the human body, inactivation of the inflammation-reducing ACE2 receptors by vaccine-generated spike proteins could have the unintended effect of materially increasing the general state of inflammation and oxidative stress in the vaccinated — even when the vaccinated person isn’t hosting a COVID19 infection. The ACE2-ACE receptor inflammation control system and how it becomes disturbed by the COVID19 virus spike proteins is described and explained at length by Verdecchia et al., 2020.1 Disturbance of this homeostasis-maintaining system by viral spike protein attachment to the ACE2 receptor sites is believed to be somewhat responsible for the relatively high mortality rates associated with the COVID19 respiratory virus.
Finally, this is entirely theoretical untested speculation, but perhaps some significant portion of the apparent immunity to infection ephemerally generated by spike protein vaccine inoculations is not the result of the desired coordinated activation of the innate and acquired immune systems, but is instead the result of simple competitive inhibition of natural COVID19 viral spikes with regard to ACE2 receptors by the competing chemical activity of vaccine-generated spike proteins. If this competition for ACE2 receptors is taking place to any great extent only in the first few months after inoculation, it may explain why immunity to infection achieved by RNA and DNA vaccines declines so very rapidly, therefore requiring repeated boosters for maintenance of this valuable infection protection through time. Under this interpretation of events, the novel COVID19 vaccines and their follow-up boosters would not be entirely acting as vaccinations ultimately inducing acquired immunity, but also (principally?) as doses of Rube Goldbergian, close imitators of generic ivermectin, HCQ, health food store zinc, or Pfizer’s new prescription wonder drug, paxlovid. For a 12/20/21 article published elsewhere and related to the subject of this paragraph, see https://anti-empire.com/its-time-for-unvaccinated-passports-house-arrest-for-the-injected/. The article discusses the observed ephemeral nature of immunity to infection of the COVID19 vaccines.
Thanks, Ann. Interesting experience in a miserable way, huh? That's the sort of thing I meant when starting to talk about the non-pharmeceutical work-arounds as the disease having a silver lining. It is, I think, making a lot of people re-examine health-related matters like diet, exercise, supplements, and modern medicine and application of governmental authority and power much closely -- and then put the results of those re-examinations into more consistent and serious practice.
Yes, you understand my postulation correctly.
I'm working on writing up summary remarks on the subject of oxidative stress and the non-pharmaceutical work-arounds right now, but one of the most straightforward studies I've found on the subject can be found here https://pubmed.ncbi.nlm.nih.gov/33680348/ . This article showed that inflammation/oxidative stress related to moderate to severe COVID19 infections could be dampened down relatively quickly with agents like melatonin, vitamin E, N-acetylcysteine (NAC), and vitamin C. This clinical study didn't cover all possible antioxidant/nutraceutical antioxidants -- for example, didn't examine vitamin A and vitamin D -- but did a great job at examining and documenting what they chose to look at.
This is fascinating, indeed. Thank you. So, if I understand you correctly, you are postulating that the spike proteins are acting as glorified doorway blockers and not necessarily acting as an immune response stimulant? Not my area of knowledge, so pardon my fumblings for comprehension. I am also fascinated with what you have to say about oxidative stress. My own run-in with Covid was purely inflammatory. Severe joint pain for days, before anosmia kicked in. Negligible pulmonary or breathing issues (only at extremity of breath a desire to cough). Interestingly, after the first forced jab, I have had chest tightness and phlegm build-up ever since, plus terrible memory lapses. I am introducing a radical change to my diet. Drastically reducing sugar, trying to eat as much raw veg of as differing colours as possible, almonds, lots of water. I haven't researched enough yet, but starting down the fumbling path of reducing inflammation and improving brain health. Also zinc, omegas, vits (but hoping to figure out their equivalents in food, too). And sunshine! Thanks again for your interesting post.